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2. 3-Phosphoinositide–Dependent Kinase 1 Potentiates Upstream Lesions on the Phosphatidylinositol 3-Kinase Pathway in Breast Carcinoma

3. Recurrent gross mutations of the PTEN tumor suppressor gene in breast cancers with deficient DSB repair

4. Lack of PTEN sequesters CHK1 and initiates genetic instability