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2. Arsenite Sensitizes Human Melanomas to Apoptosis via Tumor Necrosis Factor α-Mediated Pathway
3. Biochemical and Kinetic Characterization of the Glucocorticoid-Induced Apoptosis of Immature CD4+CD8+ Thymocytes.
4. Combined Treatment with EGFR Inhibitors and Arsenite Upregulated Apoptosis in Human EGFR-positive Melanomas: A Role of Suppression of the PI3K-AKT Pathway
5. Cooperation between STAT3 and c-Jun Suppresses Fas Transcription
6. Death receptors and melanoma resistance to apoptosis
7. Disruption of IGF-1R Signaling Increases TRAIL-Induced Apoptosis: A New Potential Therapy for the Treatment of Melanoma
8. Down-regulation of Tumor Necrosis Factor α Expression by Activating Transcription Factor 2 Increases UVC-Induced Apoptosis of Late-stage Melanoma Cells
9. Dual Treatment with COX-2 Inhibitor and Sodium Arsenite Leads to Induction of Surface Fas Ligand Expression and Fas-Ligand-mediated Apoptosis in Human Melanoma Cells
10. ERK and PI3K negatively regulate STAT-transcriptional activities in human melanoma cells: implications towards sensitization to apoptosis
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